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Endothelial dysfunction can disrupt homeostasis, increasing the risk of atherosclerotic disease and hypertension

However, mTORC1 activation only promoted metabolic reprogramming of Gclc -deficient FoB but was not sufficient to ensure antibody production, in contrast to B cell cells isolated from tuberous sclerosis complex 1 (TSC1)-deficient mice 102

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Brahma, S., Ngubo, M., Paul, S., Udugama, M

doi: 10.1038/s41467-024-51169-0 63 OjangurenI.QuirceS.BoboleaI.Prez de LlanoL.Del PozoV