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High levels of ROS activate expression of pro-inflammatory genes, with a consequent increase in adhesion molecules and release of pro-inflammatory mediators, cytokines, and chemokines [47]

[13] Published work on BPC-157 and vascular tone has discussed Src, caveolin-1, and endothelial nitric oxide synthase pathway activation in experimental systems
Singh J, Petter RC, Baillie TA, Whitty A

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