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Thus, the neuroprotective effects exhibited by curcumin in preclinical TBI models can be attributed to its ability to modulate oxidative stress, neuroinflammation, mitochondrial dysfunction, and autophagy

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Desmots, F., Rissel, M., Loyer, P., Turlin, B

Gastroenterology, 159 (3), 10851094

The precise mechanisms underlying this process are as follows: activation of the transforming growth factor-beta signaling pathway induces the phosphorylation of Smad2/3, subsequently enhancing the production of type I collagen

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