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Subsequently, we compared these results with the lipid peroxidation levels in the hippocampus and we observed a relationship between the oxidative damage and the deficit in the passive avoidance test (Graphic 2)

A0001 is therefore being developed as a treatment modality for Friedreichs ataxia patients and mitochondrial encephalomyopathy, lactic acidosis, and stroke-like episodes (MELAS) where mitochondrial redox pathologies have been identified (Hawi et al., 2012)

Lipids 1862 , 12211232 (2017)

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Apolipoprotein CIII deficiency protects against atherosclerosis in knockout rabbits

Autophagy seems to be a more competent method of damage control than HSR in the case of larger protein aggregates and dysfunctional organelles resulting after heat shock [8]