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This finding agrees with the literature whereby the role of quinone redox metabolism is an ambiguous source of toxicity, with evidence suggesting that redox cycling requires supra-pharmacological concentrations of doxorubicin to generate substantial ROS in tissues and cells 10

Instead, when an individual with mutated Fanconi anemia genes also carries the dominant-negative ALDH2*2 allele , ADH5 expression in hematopoietic tissue may be down-regulated in hematopoietic tissues, which may lead to more serious bone marrow abnormalities such as leukemia compared to patients with wild-type ALDH2*1/*1 alleles

Accordingly, this review focuses on mechanistic integration rather than exhaustive coverage of each individual domain (Panagiotou et al., 2021

doi: 10.1038/s41467-020-15136-9

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