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Functional validation using receptor-deficient models confirms that 3-adrenergic signaling is required for these effects, as disruption of this pathway attenuates peptide-induced lipolysis

Mechanistically, myocardial infarction upregulated miR-1 expression by 2.9-fold, which targeted the 3'-untranslated region of KCNJ2 (encoding Kir2.1) to reduce Kir2.1 protein levels without affecting mRNA (187)

Bioactive lipid signaling and lipidomics in macrophage polarization: impact on inflammation and immune regulation

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