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Description
Front Microbiol 14:1079653
Murine studies with specific TP53 mutants have shown that preserving either apoptotic or cell cycle regulatory functions alone is sufficient to delay tumorigenesis compared to complete TP53 loss, highlighting the distinct and complementary contributions of these two processes (119, 120)
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Implementation of pre-prescription genotyping and individualized warfarin therapy represents an opportunity to minimize the risk of haemorrhage without compromising effectiveness
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Growth hormone receptor upregulation amplifies the cellular repair response
