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However, in 2001, a handful of groundbreaking research discoveries confirmed that the Ca 2+ ionophore ionomycin could trigger an apoptosis-like cell death in erythrocytes associated with the appearance of apoptotic signs, including cell shrinkage, membrane vesiculation, or PS exposure [22, 23]

Suspected adverse reactions to any substance should be reported via the MHRA Yellow Card scheme at yellowcard.mhra.gov.uk

Regarding liver and kidney damage in MTX treatment, Bedoui stated that MTX hepatotoxicity may occur due to the depletion of folate reserves and MTX-polyglutamate formation in the liver (Reference Bedoui, Guillot and Slambarom41)

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Mitochondrial and endoplasmic reticulum calcium homeostasis and cell death

doi: 10.1080/1028415X.2022.2077031