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By understanding the metabolic pathway disruption and other pathophysiological determinants that dismantle ferroptosis defence, this may expose druggable targets to restore metabolic flux and recover ferroptosis defences in disease contexts

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Future research must also prioritize elucidating the precise molecular and biochemical mechanisms by which these therapeutics mediate their effects

Wendel A, Fausel M, Safayhi H et al (1984) A novel biologically active seleno-organic compoundII

(1997) Growth hormone treatment of abdominally obese men reduces abdominal fat mass, improves glucose and lipoprotein metabolism, and reduces diastolic blood pressure