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Tumor cells, indeed, have shown higher levels of GSH than normal cells and often are characterized by an overexpression of GSH-related enzymes and proteins that lead to a better resistance to antineoplastic treatments and, possibly, could favor chemoresistance [116,117,118]

The results showed that DSF/Cu induced distinct mitochondrial fragmentation and swollenness, while the clearance of ROS by NAC largely restored the mitochondrial morphology to a tubular and densely packed normal state (Fig

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