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Therefore, although in vitro models do not fully replicate the oxidative environment bacterial pathogens encounter in the host, the concepts outlined above provide us with a framework to begin to understand the mechanisms by which bacterial pathogens detoxify mammalian sources of ROS or other host factors triggered by ROS and repair damage to cellular targets

Elucidating how gut microbes contribute to the development of estrogen imbalance-related disorders through microbe-derived signaling molecules, metabolic pathways, or immunomodulatory mechanisms not only provides a mechanistic basis for distal microbial regulation, but also updates our understanding of the microbiomes role in female physiology and reproductive health

Am J Pathol 1969

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