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We suggest these findings reflect a compensatory upregulation of GSH in response to increasing brain oxidative stress with normal aging

It is widely accepted that tinnitus arises from maladaptive neuroplasticity in the central auditory system, which compensates for reduced peripheral input

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This can cause damage to cells and tissues, leading to chronic inflammation. The activation of the Nrf2 pathway by sulforaphane reduces oxidative stress in a clever variety of ways, including pumping out more glutathione to neutralise free radicals, thereby reducing inflammation

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