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TCDD induced hepatic inflammation by accelerating arachidonic acids metabolism to up-regulate cyclooxygenase and lipoxygenases activity to produce leukotrienes, leading to steatohepatitis (Doskey et al

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the effect of bradycardia on cardiac output is often underestimated Bradycardia directly pulls down the cardiac output, potentially causing shock

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The increased bioavailability of NO through CIT supplementation serves to optimize microvascular recruitment and the transport of glucose and amino acids, thereby fostering a conducive environment for leucine-mediated activation of the mTOR and exercise-induced metabolic adaptations (49, 82)