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59 The mechanisms of action of clinically used ASOs mainly include ribonuclease H (RNase H)-mediated degradation (Fig

Furthermore, inhibition of GSH synthesis with BSO in EVF3-infected cells shows GSH dependency for virus growth and dose-dependent stabilisation by GSH of EVF3 virions has been demonstrated by thermal stability assays 30

As proteins with a sulfydryl group are often target of oxidation (SH groups can be easily oxidized), small-molecular-weight thiols are potent scavengers

Notably, Fer-1 and Nec-1 prevented OGD-induced cell death, while inhibitor 3-MA and Z-VAD-FMK did not (Fig

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