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In particular, they have used complementary models of CFTR silencing and blockade (by the CFTR inhibitor, CFTRinh-172) in freshly isolated human umbilical vein endothelial cells (HUVECs)

Interestingly, when ghrelin is administered alone to cells coexpressing D1R and GHSR1a, no increase in cAMP accumulation is observed, while typical levels of Ca 2+ accumulation associated with GHSR1a activation occur (Jiang et al., 2006)

Voltage-dependent sodium channels such as SCN1A, SCN2A, SCN3A, SCN8A, SCN9A, and SCN1B, which encode Na v 1.1, Na v 1.2, Na v 1.3, Na v 1.6, Na v 1.7, and Na v 1, respectively, have had many different mutation sites reported for each gene, including both nonsense and missense mutations [26]

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[DOI] [PubMed] [Google Scholar] Nakashioya H, Nakano K, Watanabe N, Miyasaka N, Matsushita S, Kohsaka H (2011) Therapeutic effect of D1-like dopamine receptor antagonist on collagen-induced arthritis of mice

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