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This effect of quinolinic acid results in excessive activation of NMDA receptors accounting for the neurotoxicity of quinolinic acid

However, we showed that the increase of NO resulted in positive regulation of p53, which potentiated the antioxidant defense response mediated by NFE2L2 through direct upregulation of Peroxisome proliferator-activated receptor gamma coactivator 1-alpha (PGC-1a) [44,45]

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Vector integration site analyses showed polyclonal reconstitution without clonal dominance