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Studies have found that copper can induce neuronal cell ferroptosis, and FeCl2 and CuCl2 can both deplete GSH in mouse hippocampal neurons while enhancing ferroptosis induced by erastin, sulfasalazine (SSZ), and buthionine sulfoximine (BSO)

doi: 10.1111/bph.13702

Based on the above studies, we can speculate that regulating the hypoxic microenvironment and its associated HIF-1 upstream and downstream gene expression, such as VEGF, RIG-I and JAK/STAT3/HIF signaling pathways, may effectively reduce vascular inflammation and mitochondrial calcification in DM patients

It binds to the ghrelin receptor, which is present in the brain and other tissues, and increases the secretion of growth hormone [1][3]

These observations were attributed to impairments of the phospho-inositol/phosphate kinase B pathway

Apoptosis assays Unless otherwise stated, the cancer cells were stimulated with TNF- (10 ng/ml) plus cycloheximide (10 g/ml) for 6 h