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Metabolic reprogramming in astrocytes prevents neuronal death through a Uchl1/Pfkfb3/H4k8la positive feedback loop

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Multiple clinical studies indirectly support the role of aforementioned epigenetic modifications in chronic stress-induced HPA axis dysregulation and foetal developmental programming

reviewed the American College of Surgeons National Surgical Quality Improvement Program and found that 126 pregnant women were operated on for umbilical hernia repair in a 10-year period ( Figure 3 Thirty-one papers, including twenty-three case reports, were found in a recent literature search by Jensen et al

The tumor suppressor protein p53 increases the expression of the aspartate/glutamate transporter SLC1A3 and the arginine transporter SLC7A3 upon glutamine depletion, leading to resistance to treatments that deplete extracellular glutamine 114,115

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