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The glutamate hypothesis proposes that symptoms of schizophrenia and cognitive impairments are the result of the hypofunction of N-methyl-D-aspartate receptors (NMDARs) on -aminobutyric acid (GABA) interneurons in the cerebral cortex, resulting in overactivation of downstream glutamate signaling to the VTA and, possibly, consequent dopamine release in the mesolimbic system [4,21,22]

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[DOI] [PubMed] [Google Scholar] 15.Ogrek K., Matczuk M., Zajda J., Gutowska O., Wadych E

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doi: 10.1056/nejmoa2502081 [DOI] [PubMed] [Google Scholar] 17.Jones DW, Ferdinand KC, Taler SJ, Johnson HM, Shimbo D, Abdalla M, Altieri MM, Bansal N, Bello NA, Bress AP, et al