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The first described mechanism was in brain membranes and concerns physical properties of ethanol which allow it to directly interact with the lipid bilayer, thus triggering a direct membrane disorder (Goldstein, 1984

Our experience shows this is a particularly valuable protocol for gut-centric research, where the goal is to provide continuous support to the digestive lining

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24-OHC and 27-OHC are endogenous activators of LXR

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