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It highlights the importance of functional p53 in protecting skin cells from UVR damage, also suggesting that restoring wild-type p53 function could be a valuable strategy against skin cancer, especially non-melanoma skin cancers (NMSC)
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Overexpression of miR - 145 , miR - 149 , miR - 150 or miR - 218 suppressed the secretion of inflammatory cytokines that had been induced by CS through the inactivation of the NF - B signaling pathway [187,188,189, 192]
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After activation, these pathways enhance the transcription, protein expression, and stability of senescence-driving proteins such as P53 and P21 [21, 37, 43, 44]
The differential effects of mutant p53 alleles on advanced murine lung cancer
