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The finding of increased smoking in association with a simultaneously decline in E1G and PdG is consistent with the observation that the late luteal phase is accompanied by an increase in smoking behavior while the late follicular phase is not, as the latter is characterized by increasing estradiol but not progesterone [11]

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Another potentially worrisome trend is the increase in initiation by young adults (1824) and a possible increase in occasional smoking that may be associated with it

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Respondents are nested within cells cross-classified in birth cohorts and survey years (i.e., two social contexts)