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In conclusion, this review emphasizes the critical role of p53 as a key target in SC prevention, reinforcing the need for further research in this area to prompt the use of p53 chemopreventive agents to counteract the growing number of SC cases

Kucab JE, Phillips DH, Arlt VM

Pharmacology & Toxicology

Stroke 26 (8), 14091414

Although we use multiple overlapping models of HNSCC and recapitulate much of the HNSCC biology encountered in the clinical setting ( TP53 wild type vs mutant, HPV-associated vs HPV-independent) we are limited in our ability to precisely model human tobacco exposure in murine models with respect to both time and dose