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Proposed Mechanisms of Action The pathways by which cigarette smoking exerts opposite effects in CD and UC are hard to establish, due to the large number of components in the smoke and the diverse cellular and tissue functions that may be affected, such as the mucosal barrier, gut microbiota, immune system, microvasculature, and others

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Number of stars (*) indicates the number of criteria met for each of these three categories b Effect sizes are for oral and pharyngeal cancers combined and were included in the meta-analysis for oral cancer only We found that most of the included studies adjusted for potential confounders (35/38 for oral, 10/10 for pharyngeal and 15/16 for oesophageal cancers

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