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Retrieved July 7, 2013
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a low ATP/ADP ratio) activates AMPK, which in turn promotes catabolism and inhibits anabolism [72, 74]
Abstract The tumor suppressor p53 is a transcription factor involved in a variety of crucial cellular functions, including cell cycle arrest, DNA repair and apoptosis
In contrast, p53 S392A mutant blocks the binding of BRD7 to p53, which in turn prevents the mitochondrial translocation of p53 and inhibits the onset of ferroptosis [83]
