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The use of smokeless tobacco has also been associated with a significantly lower prevalence of canker sores

This choice dictates the entire evaluation framework

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Together, these adaptations converge to produce a stress-tolerant, clonally expanding phenotype that resists apoptosis and maintains persistent antioxidant activation under chronic cigarette exposure

The generated ROS can directly or indirectly damage cellular and sub-cellular targets, thus resulting in adverse biological consequences [22, 23, 35, 36]