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29 GIP Agonism as a Potential Therapeutic Approach for Atherosclerotic Cardiovascular Disease The cardioprotective actions of incretin-based agents, particularly the GLP-1RAs, have a bulk of supporting evidence in the form of clinical studies

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Moreover, Griffioen et al 151 reported that GLP-1R activation suppressed HR variability, and this effect was supported by the findings of the inhibition on release of both excitatory glutamatergic and inhibitory glycinergic neurotransmitters to the preganglionic parasympathetic cardiac vagal neurons

General interventions are required to protect against cytotoxic damage to beta cells (e.g