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A p53-derived apoptotic peptide derepresses p73 to cause tumor regression in vivo

While p53 exerts antioxidant effects by upregulating several antioxidant genes, such as glutathione peroxidase 1 ( Gpx1 ) and glutaredoxin 3 ( Grx3 ), or repressing the expression of dcyclooxygenase-2 ( Cox2 ) and nitric oxide synthase 2 ( Nos2 ) ( Tigar expression suppresses phosphofructokinase-1 activity and glucose 6-phosphate dehydrogenase (G6PD) activity, resulting in the shutdown of the glycolysis pathway without activating the pentose phosphate pathway, ultimately leading to testicular germ cell apoptosis and infertility ( On the other hand, p53 is sensitive to redox conditions

HepG2 cells can be killed by indolizine derivatives, which promoted p53 activation and increased its accumulation in nuclei 147

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Nature 531 , 253257 (2016)

In this study, we examined whether inflammation induction depends more on cytosolic nDNA or mtDNA, and which chemical constituents of CSE are involved