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This approach has been already tested in canine models for the treatment of Duchenne Muscular Dystrophy (Amoasii et al., 2013), in which they tested the recovery of functional CFTR in intestinal organoids obtained from CF patients, and proved the repairing of the mutation at the CFTR locus with CRISPR/Cas9 gene editing

The documentation is consistent with the diagnostic code supplied

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Overall, both surgical and medical treatments face limitations such as high recurrence rates or pronounced adverse effects, and there remains a lack of ideal long-term therapeutic options in clinical practice (9)

However, the absence of large-scale human clinical trials is a genuine limitation