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Curry , 476 F3d 326, 331 (5th Cir
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could account for developmental and neurochemical psychopathology observed after nicotine exposure[74] That is, nicotine may incorrectly mimic or duplicate endogenous signals produced by acetylcholine ACh, which an important part when synaptic formation is normal, by functioning as a nAChR agonist [78]
Serum cotinine is considered a reliable marker of both smoking intensity and exposure to tobacco smoke
Retrieved October 8, 2021
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