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Inhibition of GSH and Trx metabolism with buthionine sulfoximine (BSO) and auranofin (AUR), respectively, induced significant decreases in clonogenic survival compared to either drug alone in FaDu, Cal-27 and SCC-25 HNSCC cells in vitro and in vivo in Cal-27 xenografts

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Application: Designed for research and laboratory applications only

24 months from the date of Manufacturing

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This means that glutathione can act as a reservoir of glutamate, and changes in glutathione levels can affect glutamate signaling