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has recently demonstrated that cigarette smoke-induced inflammation is a function of TPM with low TPM concentrations activating xenobiotic and detoxification mechanisms and high TPM concentrations driving additional inflammatory response potentially triggering tissue damage [100]
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A previous study from our group investigated the association between smoking, oxidative stress and alleviation of clinical symptoms in patients with SCZ 50 , which demonstrated that smokers showed greater improvement in negative symptoms compared with nonsmokers
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