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In addition, TP53 mutation will increase structural instability and expose adhesion sequence wrapped in the hydrophobic core of p53 to protein surface, which will drive the formation of p53 aggregates [34]

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In tumors carrying mutp53, ectopic expression of SLC7A11 promotes tumor resistance to drugs that induce ferroptosis, further suggesting that mutp53 sensitizes cancer cells to ferroptosis by inhibiting SLC7A11 [79]

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Given the difficulty of capturing the complete human smoking phenotype using a murine model, we used a simpler conditioned media exposure model to ascertain the effects of smoke exposure on the interaction between HNSCC cells and PBMCs