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Damaged lysosomes are recognized by galectins, in particular galectin-3 (LGALS3) with activation of the lysosome quality control (LQC) system responsible for repairing, degrading, or replacing leaky lysosomes

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By selectively interfering with the FOXO4-p53 interaction, FOXO4-DRI has become a compound of interest in research models focused on senescent cell biology and age-associated cellular processes

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The rationale for combining these three peptides extends beyond simple additive effects to encompass true biological synergy, where coordinated activation of complementary pathways produces outcomes unattainable by individual components alone