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Colchicine mitigated the mRNA expression of the SASP factors induced by tobacco smoke condensate in endothelial cells (Fig
A p53-derived apoptotic peptide derepresses p73 to cause tumor regression in vivo
p53: Traffic cop at the crossroads of DNA repair and recombination
UVR also produces excessive amounts of reactive oxygen species (ROS) that can overwhelm the intracellular antioxidant molecules and activate signaling pathways related to inflammation and carcinogenesis [16]
Furthermore, one study has reported that maternal smoking has a greater impact on male fetuses than on female fetuses (Zaren et al., 2000)
