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Introduction Direct involvement of chemicals in the tobacco smoke with cancer has been shown since the 1970s and considered as pleiotropic carcinogens that cause DNA damage ( In this study, we will highlight genomic and molecular features, which can be used to develop approaches to predict cancer after long-term tobacco smoke exposure

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BA, PTA, PHBA, VN, DTBP and DIOP promoted the growth of R

Main results EC- and HTP-aerosols significantly augmented lung immune cell infiltrates equivalent to that achieved following CC-exposure

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