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Excitotoxicity was one of the first mechanisms of neuronal damage that was identified

Collectively, these pro-inflammatory cytokines can further exacerbate oxidative stress, creating a self-perpetuating cycle that sustains inflammation and tissue damage

This pro-angiogenic effect involves stimulation of vascular endothelial growth factor (VEGF) production and activation of endothelial cells to proliferate, migrate, and form new capillary networks

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