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Recent studies, including those carried out in the Kidney Precision Medicine Project, have shown, using single-cell RNA-Seq kidney research biopsies, the expression of the GLP-1 receptor in juxtaglomerular cells, endothelial cells, and, to a lesser extent, monocytes ( In CKD there is increased interest in the relative roles of local intrarenal macrophages versus infiltrating macrophages from the systemic circulation in promoting renal injury

In the EAE model, activated macrophages, monocytes, dendritic cells, as well as apoptotic oligodendrocytes release pathogen-associated molecular pattern or DAMP, like the high mobility group box protein-1 to the extracellular environment, and initiate an inflammatory response characterized by the activation of the inflammasome (Barclay and Shinohara, 2017

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Unfortunately, GLP-1 is rapidly degraded by the enzyme dipeptidyl peptidase-4 (DPP-4), which limits the therapeutic efficacy of native GLP-1 (Deacon et al., 1995)

Neither shared a full-blown recovery story