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Factors such as the accumulation of lipid metabolites, inflammatory processes, and dysfunction in hypothalamic neurons may also contribute to obesity, potentially explaining the biological mechanisms that favor the maintenance of excessive body fat [7]

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This viewpoint promotes a transition from viewing GLP-1 RAs in a disease-specific manner to a more comprehensive understanding of whether these drugs impact various aging hallmark processes, including mitochondrial dysfunction, impaired proteostasis, and the decline in stem and progenitor cell functionelements that contribute to the diminished functional reserve seen with aging

Genetic data also informs testosterone dosing optimization in some cases, though testosterone pharmacogenomics is distinct from peptide pathway testing