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Various GLP-1RAs have been demonstrated to have significant benefits in in vitro and in vivo models of neurodegenerative diseases through modulating a variety of pathogenic mechanisms, including neuroinflammation, autophagy, mitochondrial dysfunction, and the abnormal phosphorylation of pathognomonic proteins

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Here, we confirm that neither Epsin nor the cytosolic domains of DSL proteins are required for Notch signaling in C

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Not approved for human or veterinary use.