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In addition, the gut-derived metabolite imidazole propionate has been shown to drive -synuclein pathology in preclinical and patient-derived PD models, providing a direct link between dysbiosis and -synuclein aggregation (103)

Advanced hepatic disease/hepatic functional impairment

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1), and also acts on receptors in pancreatic -cells, myocytes in the sinoatrial node, and Brunners glands in the duodenum(Knudsen et al 2014)

doi: 10.1038/ijo.2010.59 63 GaultV

F.VaduganathanM.RosenstockJ.et al (2020)