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This is important because GLP-1 drugs were originally designed to have a long half-life in blood to maximise their efficacy in the treatment of diabetes which prevents them from entering the brain (Hlscher)

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10.1016/j.molmet.2018.02.008 78

But in practice, these two compounds produce completely different outcomes, work through fundamentally different mechanisms, and carry very different risk profiles

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They bind to highly selective specific receptors, GLP-1 has a very low affinity towards the GIP receptor and vice versa (43)