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[DOI] [PMC free article] [PubMed] [Google Scholar] Rudinsky A.J., Adin C.A., Borin-Crivellenti S., Rajala-Schultz P., Hall M.J., Gilor C

This predisposition is likely multifactorial, involving shared genetic, inflammatory, and neuroendocrine mechanisms [61]

This variability could have impacted the consistency of the results and the overall interpretation of gastrointestinal adverse events

T2DM was defined as a fasting plasma glucose level 126 mg/dL or assigned International Classification of Diseases 10th Revision (ICD-10) codes E11E14 with taking glucose-lowering drugs or insulin

This was evident when comparing exenatide and semaglutide chromatographic profiles: exenatide, a non-lipidated peptide with several acidic residues, was strongly affected by NSA (likely due to ionic and dipole-ionic interactions with metal surfaces), leading to decreased efficiency