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Nonetheless, in line with a role for PDX1 in age-related changes in GIP secretion, suppression of PDX1 using intestine-specific gene transfer in mice reduces K-cell number, Gip expression and GIP content in the small intestine [192]

A study reported that chronic low-grade ER stress in the livers of obese MASLD mice stimulates gluconeogenesis by stabilizing cyclic AMP-responsive element-binding protein (CREB) through ubiquitin-specific peptidase 14 (USP14) activation (149)

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