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Zhang T, Cheng JK, Hu YM

Its dual GIP/GLP-1 mechanism targets appetite suppression and insulin response, but does not add the energy-expenditure dimension that retatrutide's glucagon arm provides

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Hyperinsulinemia and insulin resistance promote tumor growth through various mechanisms: directly by activating the insulin receptor signaling pathway and indirectly by stimulating the insulin-like growth factor (IGF) receptor system, which in turn fosters cellular proliferation, migration, and invasion while inhibiting programmed cell death [11]