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Assuming a preferential central action of GLP1/E, we conducted label-free, quantitative proteomic analyses in the hypothalamus of the two models of PCOS, after chronic treatment with the di-agonist, as a means to disclose putative pathways for the metabolic actions of the di-agonist, and the basis for the partially differential responses between the two models of PCOS, which diverge also in terms of phenotypic presentation

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[DOI] [PMC free article] [PubMed] [Google Scholar] 61.An Endogenous GLP-1 Circuit Engages VTA GABA Neurons to Regulate Mesolimbic Dopamine Neurons and Attenuate Cocaine Seeking

doi: 10.1056/NEJMp1314078 [DOI] [PubMed] [Google Scholar] 78

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