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According to Lilly, Plaintiffs have already sought discovery concerning conditions such as malnutrition, dehydration, pulmonary aspiration, Wernickes encephalopathy, pancreatitis, and acute kidney injury

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GLP-1 acts on insulin secretion and satiety: it increases insulin and decreases glucagon secretion, thereby blocking the endogenous production of glucose and decreasing glycemia after a meal
However, in rats, GLP-1 elevation of BP is preserved upon pretreatment with reserpine, propanolol or phentolamine, thus indicating that the hypertensive effect of GLP-1 does not exclusively depend on catecholamine signaling [634]
