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Additional studies demonstrated that SREBP-1c -/- mice had impaired induction of hepatic genes coding for fatty acid biosynthesis (e.g., acetyl-CoA carboxylase, fatty acid synthase, and stearoyl-CoA desaturase) compared to wild type mice [17]
Therefore, therapies capable of producing sustained weight reduction may substantially alter disease progression rather than simply treating elevated glucose levels
However, the substitution of residues in the N-terminal half of GLP-1 for those of glucagon had much smaller effects (Hjorth et al ., 1994
