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12 Oxidative stress, 13 mitochondrial dysfunction, 14 and endoplasmic reticulum (ER) stress 15 are all involved in NDs induced by brain IR

Perhaps the agonist-induced interaction with arrestins at the membrane is too short-lived to confer significant GLP-1R desensitization, due to the rapid caveolin-mediated GLP-1R internalization upon agonist activation

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