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Treatment of type 2 diabetes with incretin-based therapies
Am Surg
[DOI] [PubMed] [Google Scholar] 236.Orskov C., Wettergren A., Holst J.J
DB enhances insulin release through mechanisms involving KATP channels, bitter taste receptor signaling, and intraislet GLP-1 secretion
In vitro investigations revealed its capacity to downregulate inflammatory gene expression and suppress the synthesis of interleukin 6 (IL-6), prostaglandin E2 and nitric oxide in chondrocytes and macrophages in a dose-dependent manner
When traveling or commuting, keeping medication in that safe zone prevents potency loss and reduces injection discomfort
